Carcass Transportation Regulations in the United States and Canada

cwd_mapDownload the full Chronic Wasting Disease and Cervidae Regulations in North America. [PDF]

The number one objective in the management of CWD is to prevent its spread into new areas. One theoretical mode of disease transmission is via infected carcasses. Therefore, in an effort to minimize the risk of disease spread, a number of states have adopted regulations affecting the transportation of hunter-harvested deer and elk.

Since the suspected infective agent (prion) is concentrated in the brain, spinal cord and lymph glands, the most common regulation is the prohibition of the importation of whole carcasses harvested from CWD areas. Some states, like Colorado, also have established regulations addressing the transport of deer and elk out of CWD areas. Generally, states that have adopted carcass transportation regulations do not allow the importation of any brain or spinal column tissue and allow transport of only the following:

  • Meat that is cut and wrapped (either commercially or privately).
  • Quarters or other portions of meat with no part of the spinal column or head attached.
  • Meat that has been boned out.
  • Hides with no heads attached.
  • Clean (no meat or tissue attached) skull plates with antlers attached.
  • Antlers with no meat or tissue attached.
  • Upper canine teeth, also known as "buglers," "whistlers," or "ivories."
  • Finished taxidermy.

A summary of state-by-state carcass transportation regulations is provided in Column J of the regulations on each state page (accessible from the home page) or on the map. Since these regulations are continually evolving, it is recommended that before hunting you check the CWD regulations in your home state, the state in which you will be hunting and states in which you will travel through en route home from your hunting area. Most state wildlife agencies provide regulations information on their websites, and may be accessed via the clickable map on the home page.

The Carcass Transport and Disposal Working Group of the Association of Fish and Wildlife Agencies (AFWA) Fish and Wildlife Health Committee developed the following guidelines for regulatory and non-regulatory approaches to carcass transport and disposal. The intent of the working group is to encourage states to adopt policies that minimize risk; do not hinder hunting, wild cervid population management, or disease control; are easily understood; and promote compliance because they are consistent and well-justified. The recommendations are based on current knowledge of CWD and may be updated when new information becomes available. The Working Group recognizes state wildlife management agencies will tailor their approach to fit individual concerns and situations, and asks that agency directors, through AFWA, give serious and urgent consideration to this matter so that this potential risk of CWD spread can be minimized.

Transport and Disposal of Hunter-killed Cervid Carcasses: Recommendations to Wildlife Agencies to Reduce Chronic Wasting Disease Risks [PDF]

US Legislation

---April 6, 2004---
Senate Hearing on S1366 - Chronic Wasting Disease Financial Assistance Act of 2003

Senate Environment and Public Works Subcommittee on Fisheries, Wildlife and Water
April 6, 2004

CWD Alliances’ Testimony PDF document
Other Testimony

---January 9, 2004---
S 2007 - BSE and Other Prion Disease Prevention and Public Health Protection Act

To provide better protection against bovine spongiform encephalopathy and other prion diseases.
S 2007 PDF document
S 2007 Word document

---June 19, 2003---
Congressional Hearing on HR 2057

U.S. House of Representatives
House Resources Committee
Subcommittees on Forests and Forest Health, and
Fisheries Conservation, Wildlife and Oceans
Thursday, June 19, 2003

Testimony

---June 9, 2003---
HR 2431 - Chronic Wasting Disease Task Force Establishment Act of 2003 (Introduced in House)

To establish a National Chronic Wasting Disease Task Force, and for other purposes.
HR2431 Word document

---June 9, 2003---
HR 2430 - Chronic Wasting Disease Research, Monitoring, and Education Enhancement Act of 2003 (Introduced in House)

To amend the Fish and Wildlife Coordination Act to coordinate and strengthen scientific research and monitoring, and to promote public outreach, education, and awareness, of Chronic Wasting Disease affecting free-ranging populations of deer and elk, and for other purposes.
HR2430 Word document

---June 9, 2003---
S 1366 - Chronic Wasting Disease Financial Assistance Act of 2003 (Introduced in Senate)

To authorize the Secretary of the Interior to make grants to State and tribal governments to assist State and tribal efforts to manage and control the spread of chronic wasting disease in deer and elk herds, and for other purposes.
S1366 PDF document | Word document

---June 9, 2003---
HR 2636 - Chronic Wasting Disease Financial Assistance Act of 2003 (Introduced in House)

To authorize the Secretary of the Interior to make grants to State and tribal governments to assist State and tribal efforts to manage and control the spread of chronic wasting disease in deer and elk herds, and for other purposes.
HR2636 PDF document | Word document

---May 9, 2003---
S 1036 - Chronic Wasting Disease Support Act of 2003

Introduce in the Senate May 9, 2003 by Senator Allard (CO)
S1036 Word document
S1036 PDF document

---May 9, 2003---
HR 2057 - Chronic Wasting Disease Support for States Act of 2003

Introduced in the House of Representatives May 9, 2003 by Rep. McInnis (CO)
HR2057 Word document
HR2057 PDF document

---April 18, 2003---
FY 2004 Budget - Conservation Organizations Request Congressional Support for CWD

24 organizations sign letter requesting funding for National CWD Plan- April 18, 2003
Letter Word document

---May 16, 2002---
Congressional Hearing on Chronic Wasting Disease

U.S. House of Representatives
House Resources Committee
Subcommittees on Forests and Forest Health, and
Fisheries Conservation, Wildlife and Oceans
May 16, 2002
CWD Alliances’ Testimony PDF document | Word document

  • All
  • 2
  • Recent News
  • Default
  • Title
  • Date
  • Random
load more hold SHIFT key to load all load all

News by Year

2017 (5)2016 (1)2015 (2)2014 (2)2012 (2)2011 (4)2010 (3)2009 (5)2008 (4)2007 (8)2006 (21)2005 (19)2004 (26)2003 (45)2002 (25)

Category Archives: National News

Chronic Wasting Disease Update

CWD Update 84 January 27, 2007

State and Provincial Updates

Alberta: Alberta is continuing testing for the provincial chronic wasting disease (CWD) surveillance program for 2006-07. One additional case of CWD in wild deer was confirmed on January 2. This brings the total to 17 cases in wild deer in Alberta since the first case in September 2005. The most recent case was taken by a hunter near Chauvin, not far from an earlier case reported in late December.

Alberta Sustainable Resource Development’s CWD information is at: www.srd.gov.ab.ca/fw/diseases/CWD. A map showing the locations of Alberta CWD+ animals is at: http://www.srd.gov.ab.ca/fw/diseases/CWD/pdf/CWD_positive_Dec2006.pdf.

West Virginia: On December 22, 2006 the West Virginia DNR announced that a hunter-harvested deer taken during the 2006 hunting season tested positive for CWD. The 2.5 year old buck, one of 1355 deer sampled in Hampshire County during the 2006 hunting season, was taken in close proximity to the previously-established cluster of CWD+ deer in Hampshire County. CWD has now been detected in a total of 10 deer in West Virginia, all in Hampshire County: one road-killed deer, four deer collected by the DNR in 2005, four deer collected by the DNR in 2006 and one hunter-harvested deer during the 2006 deer season.

West Virginia DNR CWD information is at: http://www.wvdnr.gov/hunting/ChronicWaste.shtm. A WVDNR fact sheet is at: http://www.wvdnr.gov/hunting/PDFFiles/CWDfactsheet.pdf.

Wisconsin: The Wisconsin DNR has begun agency sharpshooting at Devil’s Lake State Park and the Southern Unit of Kettle Moraine State Forest. Devil’s Lake S.P. is the furthest north that CWD has been detected in the state; three CWD+ deer have been detected in the 10,200 acre park in Sauk County in the last year. Kettle Moraine State Forest lies just north of the southeastern CWD zone in Wisconsin; five hunter-harvested deer from the 6400 acre Walworth County portion of the forest tested positive this year. In an additional note, the DNR has begun conducting this year’s winter helicopter deer counts in CWD-affected areas. Recent snowfall and cold temperatures are providing good conditions for these efforts to determine deer density within Wisconsin’s CWD zones.

Wisconsin Department of Natural Resources CWD information, including an interactive map showing locations of positives and overall surveillance effort, is at: http://www.dnr.state.wi.us/org/land/wildlife/whealth/issues/CWD/index.htm.

Wyoming: The Wyoming Game and Fish Department has finished its fourth year of comprehensive Chronic Wasting Disease surveillance and added two deer hunt areas and two elk hunt areas to its list of areas where CWD has been detected. Game and Fish personnel collected 4,653 deer, elk and moose samples in 2006. Of those, 116 animals tested positive for CWD – 88 mule deer, 13 white-tailed deer and 15 elk. New cases of CWD were diagnosed in deer hunt area 4 east of Sundance, deer hunt area 11 in Niobrara and Weston counties and elk hunt areas 16 and 22 in northern Carbon County.

The Wyoming Game & Fish Department 2006 surveillance summary press release is at: http://gf.state.wy.us/services/news/pressreleases/06/12/15/061215_2.asp. Wyoming Game & Fish Department CWD Information is at: http://gf.state.wy.us/services/education/cwd/index.asp.

Recent Publications

Production of cattle lacking prion protein. Jürgen A Richt, Poothappillai Kasinathan, Amir N Hamir, Joaquin Castilla, Thillai Sathiyaseelan, Francisco Vargas, Janaki Sathiyaseelan, Hua Wu, Hiroaki Matsushita, Julie Koster, Shinichiro Kato, Isao Ishida, Claudio Soto, James M Robl & Yoshimi Kuroiwa Nature Biotechnology 25, 132-138 (2006).

Abstract: Prion diseases are caused by propagation of misfolded forms of the normal cellular prion protein PrPC, such as PrPBSE in bovine spongiform encephalopathy (BSE) in cattle and PrPCJD in Creutzfeldt-Jakob disease (CJD) in humans. Disruption of PrPC expression in mice, a species that does not naturally contract prion diseases, results in no apparent developmental abnormalities. However, the impact of ablating PrPC function in natural host species of prion diseases is unknown. Here we report the generation and characterization of PrPC-deficient cattle produced by a sequential gene-targeting system. At over 20 months of age, the cattle are clinically, physiologically, histopathologically, immunologically and reproductively normal. Brain tissue homogenates are resistant to prion propagation in vitro as assessed by protein misfolding cyclic amplification. PrPC-deficient cattle may be a useful model for prion research and could provide industrial bovine products free of prion proteins. http://www.nature.com/nbt/journal/v25/n1/abs/nbt1271.html


The role of the cellular prion protein in the immune system. J. D. Isaacs, G. S. Jackson, and D. M. Altmann Clinical & Experimental Immunology 146 (1), 1–8 (2006).

Abstract: Prion protein (PrP) plays a key role in the pathogenesis of prion diseases. However, the normal function of the protein remains unclear. The cellular isoform (PrPC) is expressed widely in the immune system, in haematopoietic stem cells and mature lymphoid and myeloid compartments in addition to cells of the central nervous system. It is up-regulated in T cell activation and may be expressed at higher levels by specialized classes of lymphocyte. Furthermore, antibody cross-linking of surface PrP modulates T cell activation and leads to rearrangements of lipid raft constituents and increased phosphorylation of signalling proteins. These findings appear to indicate an important but, as yet, ill-defined role in T cell function. Although PrP-/- mice have been reported to have only minor alterations in immune function, recent work has suggested that PrP is required for self-renewal of haematopoietic stem cells. Here, we consider the evidence for a distinctive role for PrPC in the immune system and what the effects of anti-prion therapeutics may be on immune function. http://www.blackwell-synergy.com/doi/abs/10.1111/j.1365-2249.2006.03194.x.


Prions and their partners in crime. Byron Caughey and Gerald S. Baron Nature 443, 803-810 (19 October 2006)

Abstract: Prions, the infectious agents of transmissible spongiform encephalopathies (TSEs), have defied full characterization for decades. The dogma has been that prions lack nucleic acids and are composed of a pathological, self-inducing form of the host’s prion protein (PrP). Recent progress in propagating TSE infectivity in cell-free systems has effectively ruled out the involvement of foreign nucleic acids. However, host-derived nucleic acids or other non-PrP molecules seem to be crucial. Interactions between TSE-associated PrP and its normal counterpart are also pathalogically important, so the physiological functions of normal PrP and how they might be corrupted by TSE infections have been the subject of recent research. http://www.nature.com/nature/journal/v443/n7113/abs/nature05294.html.


The following review article, authored by Beth Williams, was published in 2005, following her unfortunate and untimely death. While aspects of our knowledge regarding CWD have been updated by more recent science, this review article is probably the most thorough ever written and is highly recommended to bolster the reader’s overall understanding of CWD.

Chronic Wasting Disease. E. S. Williams Veterinary Pathology 42:530–549 (2005)

Abstract: Chronic wasting disease (CWD) is a unique transmissible spongiform encephalopathy (TSE) of mule deer (Odocoileus hemionus), white-tailed deer (O. virginianus), and Rocky Mountain elk (Cervus elaphus nelsoni). The natural history of CWD is incompletely understood, but it differs from scrapie and bovine spongiform encephalopathy (BSE) by virtue of its occurrence in nondomestic and free-ranging species. CWD has many features in common with scrapie, including early widespread distribution of disease-associated prion protein (PrPd) in lymphoid tissues, with later involvement of central nervous system (CNS) and peripheral tissues. This distribution likely contributes to apparent efficiency of horizontal transmission and, in this, is similar to scrapie and differs from BSE. Clinical features and lesions of CWD are qualitatively similar to the other animal TSEs. Microscopically, marked spongiform lesions occur in the central nervous system (CNS) after a prolonged incubation period and variable course of clinical disease. During incubation, PrPd can be identified in tissues by antibody-based detection systems. Although CWD can be transmitted by intracerebral inoculation to cattle, sheep, and goats, ongoing studies have not demonstrated that domestic livestock are susceptible via oral exposure, the presumed natural route of exposure to TSEs. Surveillance efforts for CWD in captive and free-ranging cervids will continue in concert with similar activities for scrapie and BSE. Eradication of CWD in farmed cervids is the goal of state, federal, and industry programs, but eradication of CWD from free-ranging populations of cervids is unlikely with currently available management techniques. http://www.vetpathology.org/cgi/content/abstract/42/5/530.

Upcoming Conferences

Transmissible Spongiform Encephalopathies: The Definitive American TSE Meeting February 12-13, 2007, Baltimore, Maryland http://www.healthtech.com/2007/tse/index.asp

Chronic Wasting Disease Update

CWD Update 82 November 20, 2006

State Updates

New York: John Major, New York Department of Environmental Conservation (DEC), provides the following: For 2006, Chronic Wasting Disease regulations in New York have been updated in response to the finding of the disease in a moose in Colorado and deer in West Virginia. Hunters bringing carcasses into New York from West Virginia must now process them to remove tissues of concern prior to import, and moose have been added to the list of susceptible species that previously included deer and elk. Last year, more than 8,000 wild deer were tested for CWD in New York following the discovery of CWD in 2 wild and 5 captive deer in April 2005, and no additional animals have tested positive for the disease. Special restrictions are in place within the CWD Containment Area of Oneida and Madison counties governing how harvested deer and specific deer parts may be possessed, transported, and disposed. Mandatory testing of all deer taken in the Containment Area will be occurring again this fall, as well as random testing in other counties throughout New York State. Surveillance will continue at the same level this year. As of November 17, more than 3,200 samples (including more than 800 from the Containment Area) have been tested this year, none have detected CWD. The NY DEC maintains CWD information at: http://www.dec.state.ny.us/website/dfwmr/wildlife/deer/cwd.html.

Wisconsin: The Wisconsin DNR has been diligently working to manage CWD since it was detected in free-ranging deer in 2002. In September, DNR staff held a retreat to discuss their progress, relevant science, and future management options. On October 25, WDNR Secretary Scott Hassett presented a memo to the WI DNR Board, outlining the CWD situation in Wisconsin. Secretary Hassett’s memo can be viewed at: http://www.dnr.state.wi.us/org/land/wildlife/whealth/issues/cwd/doc/Hasset_CWDMgmt.pdf.

Also in Wisconsin, the Legislative Audit Bureau recently completed an audit of Wisconsin’s CWD program. The results of the audit (98 page report) can be obtained at: http://www.legis.state.wi.us/lab/reports/06-13Full.pdf. The four page summary report is at: http://www.legis.state.wi.us/lab/reports/06-13Full.pdf. Response from the Wisconsin DNR, including their formal response and press release are at: http://www.dnr.state.wi.us/org/land/wildlife/whealth/issues/CWD/index.htm.

Wyoming: A male white-tailed deer killed in Hunt Area 4, east of Sundance in the Moskee area on November 4, 2006 has tested positive for CWD. Hunt Area 4 borders the Wyoming/South Dakota state line. This is the first CWD-positive animal reported in this hunt area. Press Release available at: http://gf.state.wy.us/services/news/pressreleases/06/11/17/061117_3.asp Wyoming G&F CWD information is at: http://gf.state.wy.us/services/education/cwd/index.asp.

Northeast Association of Fish and Wildlife Agencies: On November 2, 2006, the Northeast Association adopted a regional CWD Plan. Here (PDF) is a link to the document.


Recent Publications

Infectious agent of sheep scrapie may persist in the environment for at least 16 years. Gudmundur Georgsson, Sigurdur Sigurdarson and Paul Brown J Gen Virol 87 (2006), 3737-3740; DOI 10.1099/vir.0.82011-0

Abstract: In 1978, a rigorous programme was implemented to stop the spread of, and subsequently eradicate, sheep scrapie in Iceland. Affected flocks were culled, premises were disinfected and, after 2–3 years, restocked with lambs from scrapie-free areas. Between 1978 and 2004, scrapie recurred on 33 farms. Nine of these recurrences occurred 14–21 years after culling, apparently as the result of environmental contamination, but outside entry could not always be absolutely excluded. Of special interest was one farm with a small, completely self-contained flock where scrapie recurred 18 years after culling, 2 years after some lambs had been housed in an old sheep-house that had never been disinfected. Epidemiological investigation established with near certitude that the disease had not been introduced from the outside and it is concluded that the agent may have persisted in the old sheep-house for at least 16 years. http://vir.sgmjournals.org/cgi/content/abstract/87/12/3737


A blood test for prion: disease associated prion aggregate is detected in the blood of infected but asymptomatic animals. Binggong Chang, Xin Cheng, Shaoman Yin, Tao Pan, Hongtao Zhang, Poki Wong, Shin-Chung Kang, Fan Xiao, Huimin Yan, Chaoyang Li, Lisa L. Wolfe, Michael W. Miller, Thomas Wisniewski, Mark I. Greene, and Man-Sun Sy Clin. Vaccine Immunol. doi:10.1128/CVI.00341-06

Abstract: We have developed a sensitive in vitro assay for detecting disease associated prion aggregates by combining an aggregation specific enzyme-linked immunosorbent assay (AS-ELISA) with a Fluorescent Amplification Catalyzed by T7 RNA polymerase Technique (FACTT). The new assay, named AS-FACTT, is much more sensitive than AS-ELISA and could detect prion aggregates in the brain of mice as early as 7 days after an intra-peritoneal inoculation of PrPSc. However, AS-FACTT was still unable to detect prion aggregates in blood of infected mice. To further improve the detection limit of AS-FACTT, we added an additional prion amplification step (Am) and developed a third generation assay, termed Am-A-FACTT. Am-A-FACTT has 100% sensitivity and specificity in detecting disease-associated prion aggregates in blood of infected mice at late but still asymptomatic stages of disease. At a very early stage, Am-A-FACTT had a sensitivity of 50% and specificity of 100%. Most importantly, Am-A-FACTT also detects prion aggregates in blood of mule deer infected with a naturally occurring prion disease, chronic wasting disease. Application of this assay to cattle, sheep, and humans could safeguard food supplies and prevent human contagion. http://cvi.asm.org/cgi/content/abstract/CVI.00341-06v1


Spatial Epidemiology of Chronic Wasting Disease in Wisconsin White-Tailed Deer. Damien O. Joly, Michael D. Samuel, Julia A. Langenberg, Julie A. Blanchong, Carl A. Batha, Robert E. Rolley, Delwyn P. Keane and Christine A. Ribic Journal of Wildlife Diseases, 42(3), 2006, pp. 578-588

Abstract: Chronic wasting disease (CWD) is a fatal, emerging disease of cervids associated with transmissible protease-resistant prion proteins. The potential for CWD to cause dramatic declines in deer and elk populations and perceived human health risks associated with consuming CWD-contaminated venison have led wildlife agencies to embark on extensive CWD control programs, typically involving culling to reduce deer populations. We characterized the spatial distribution of CWD in white-tailed deer (Odocoileus virginianus) in Wisconsin to facilitate CWD management. We found that CWD prevalence declined with distance from a central location, was locally correlated at a scale of 3.6 km, and was correlated with deer habitat abundance. The latter result is consistent with patterns expected for a positive relationship between density and prevalence of CWD. We recommend management activities focused on culling in geographic areas with high prevalence to have the greatest probability of removing infected individuals. Further research is needed to elucidate the factors involved in CWD spread and infection rates, especially the role of density-dependent transmission. http://www.jwildlifedis.org/cgi/content/abstract/42/3/578


White-Tailed Deer Harvest from the Chronic Wasting Disease Eradication Zone in South-Central Wisconsin. Julie A. Blanchong, Damien O. Joly, Michael D. Samuel, Julia A. Langenberg, Robert E. Rolley and Janet F. Sausen Wildlife Society Bulletin 34(3):725-731

Abstract: Chronic wasting disease (CWD) was discovered in free-ranging white-tailed deer (Odocoileus virginianus) in south-central Wisconsin in 2002. The current control method for CWD in the state is the harvest of deer from affected areas to reduce population density and lower CWD transmission. We used spatial regression methods to identify factors associated with deer harvest across south-central Wisconsin. Harvest of deer by hunters was positively related to deer density (slope¼0.003, 95%CI¼0.0001–0.006), the number of landowners that requested harvest permits (slope¼0.071, 95% CI¼0.037–0.105), and proximity to the area of highest CWD infection (slope¼_0.041, 95% CI¼_0.056–_0.027). Concomitantly, harvest was not impacted in areas where landowners signed a petition protesting intensive deer reduction (slope ¼_0.00006, 95% CI ¼_0.0005–0.0003). Our results suggest that the success of programs designed to reduce deer populations for disease control or to reduce overabundance in Wisconsin are dependent on landowner and hunter participation. We recommend that programs or actions implemented to eradicate or mitigate the spread of CWD should monitor and assess deer population reduction and evaluate factors affecting program success to improve methods to meet management goals.


Passage of chronic wasting disease prion into transgenic mice expressing Rocky Mountain elk (Cervus elaphus nelsoni) PrPC. Giuseppe LaFauci, Richard I. Carp, Harry C. Meeker, Xuemin Ye, Jae I. Kim, Michael Natelli, Marisol Cedeno, Robert B. Petersen, Richard Kascsak and Richard Rubenstein J Gen Virol 87 (2006), 3773-3780; DOI 10.1099/vir.0.82137-0

Abstract: Chronic wasting disease (CWD) of elk (Cervus elaphus nelsoni) and mule deer (Odocoileus hemionus) is one of three naturally occurring forms of prion disease, the others being Creutzfeldt–Jakob disease in humans and scrapie in sheep. In the last few decades, CWD has spread among captive and free-ranging cervids in 13 US states, two Canadian provinces and recently in Korea. The origin of the CWD agent(s) in cervids is not known. This study describes the development of a transgenic mouse line (TgElk) homozygous for a transgene array encoding the elk prion protein (PrPC) and its use in propagating and simulating CWD in mice. Intracerebral injection of one mule deer and three elk CWD isolates into TgElk mice led to disease with incubation periods of 127 and 95 days, respectively. Upon secondary passage, the incubation time was reduced to 108 and 90 days, respectively. Upon passage into TgElk mice, CWD prions (PrPSc) maintained the characteristic Western blot profiles seen in CWD-affected mule deer and elk and produced histopathological modifications consistent with those observed in the natural disease. The short incubation time observed on passage from cervid to mouse with both mule deer and elk CWD brain homogenates and the demonstrated capacity of the animals to propagate (mouse to mouse) CWD agents make the TgElk line a valuable model to study CWD agents in cervid populations. In addition, these results with this new transgenic line suggest the intriguing hypothesis that there could be more than one strain of CWD agent in cervids. http://vir.sgmjournals.org/cgi/content/abstract/87/12/3773


Upcoming Conferences:

The 67th Midwest Fish and Wildlife Conference December 3-6, 2006 Omaha, Nebraska http://www.ngpc.state.ne.us/midwest2006 A special CWD session will be held on Tuesday, December 5. Eleven papers on CWD will be presented. Titles and abstracts are available on the conference web site. Click on “Program @ Glance” then “CWD Symposium.”

Transmissible Spongiform Encephalopathies: The Definitive American TSE Meeting February 12-13, 2007 Baltimore, Maryland http://www.healthtech.com/2007/tse/index.asp The conference agenda can be downloaded from the conference web site. Early registration closes December 1.

Chronic Wasting Disease Update

CWD UPDATE 81 November 14, 2006

USDA-APHIS-Veterinary Services has published a request for comments on the three petitions received from the Association of Fish and Wildlife Agencies, the U. S. Animal Health Association, and the National Assembly of State Animal Health Officials protesting portions of their rule on the interstate transportation of captive cervids and the federal herd-monitoring program. The notice was in the Federal Register of November 3, 2006 and can be accessed here. It is very important that all interested parties review this notice and submit comments. The two main issues are the preemption of state authority and allowing movement across state lines after only one year of monitoring, gradually increasing to 5 years.

An elk killed recently in the Shirley Mountains northwest of Medicine Bow, Wyoming has tested positive for chronic wasting disease. The hunter-killed elk comes from Hunt Area 16 where CWD had been detected in deer previously, although this is the first elk from that hunt unit to test positive. Elk in two other elk hunt areas immediately to the east and south of Hunt Area 16 have tested positive for the disease in past years. The Colorado Division of Wildlife reports finding two additional positive moose. The two bull moose were harvested legally in northern Colorado near Glendevey recently. One of the moose was from the same herd that the first positive moose was from. This brings the total wild moose positive for CWD to 3 out of 528 tested in Colorado.

Results for CWD testing in South Dakota from July 1 to November 13, 2006 have been reported by South Dakota Game, Fish, and Parks. The samples tested and results are; 444 elk sampled–438 results returned as NOT Positive–4 results pending (2 POSITIVE ELK FOUND), 111 mule deer sampled–26 results returned as NOT Positive–85 results pending, 402 white-tailed deer–44 results returned as NOT Positive– 358 results pending The two positive elk were both female and hunter harvested. Both were from Custer County, one being taken in Custer State Park. To date, South Dakota has found 49 cases of CWD (32 deer and 17 elk) in free ranging deer and elk since testing began in 1997. Wind Cave National Park accounts for 16 of these animals (8 elk, 8 deer). A total of 13,262 wild deer and elk have been tested for CWD since 1997.

Wisconsin DNR reports on results to date for this years testing. During the fall hunting season (still in progress), they have collected 5,429 samples and received results from 3,456 of these. There were 6 positive in this number, 4 from the disease eradication zone and 2 from the herd reduction zone. This brings their totals to 105,594 animals sampled with 657 positive.

The New Mexico Game and Fish Department reports that they have detected CWD in one mule deer on the Stallion site of White Sands Missile Range. This location is approximately 100 miles north of the main infection area near the Missile Range headquarters and 75 miles north of the focus of infection in the southern Sacramento Mountains. The Wyoming Game and Fish Department reports that a white-tailed deer harvested in Hunt Area 9 near Newcastle has tested positive for chronic wasting disease. The disease had not previously been found in deer Hunt Area 9. The deer was harvested on a ranch southeast of Newcastle in early October. The location is approximately 18 miles from where the disease has been found in South Dakota in the same drainage.

OK, this is it, my very last and final CWD update. My last day of work is next Wednesday, November 22, 2006. I will be enjoying the mountains of northern New Mexico and trying my hand at fishing and hunting again. I have been blessed by my association with the brightest and best in our business and will miss the friendships and camaraderie from times past. Bryan Richards, CWD Project Leader for the National Wildlife Health Lab in Madison, Wisconsin has agreed to take over the responsibility of issuing these updates. Lets all help Bryan out by providing him with information on what is going on in your neck of the woods as concerns CWD so he can get the information out to others. Bryan’s email is [email protected]. If you need to talk to Bryan, he can be reached at 608-270-2485. Thanks Bryan for taking on this job, and thanks to each and every one reading this for their help and friendship during these interesting years. I will plan on keeping in touch through the updates and will be doing a little consulting, so might see some of you at future meetings (if it doesn’t interfere with my hunting and fishing). Goodbye and God Bless.

USDA Seeks Comments on Three Petitions Regarding Delayed Implementation of Chronic Wasting Disease Regulations

WASHINGTON, Nov. 2, 2006–The U.S. Department of Agriculture’s Animal and Plant Health Inspection Service is soliciting comments on three petitions requesting that APHIS reconsider provisions and delay a recent final rule establishing a herd certification program and interstate movement restrictions for cervids to control the spread of chronic wasting disease (CWD). The CWD herd certification program and interstate movement of farmed or captive deer, elk and moose final rule, published on July 21, originally had an effective date of Oct. 19. However, in early August, APHIS received petitions from the Association of Fish and Wildlife Agencies, the National Assembly of State Animal Health Officials and the United States Animal Health Association asking for a delay and review. APHIS is taking this opportunity to solicit comments on the merits of these three petitions.

While the public is invited to comment on any of the issues raised by the petitions, APHIS is particularly interested in receiving comments related to the following areas:

  • Consider the alternatives of implementing a federal interstate movement standard versus allowing individual state standards to apply? What hardships or benefits would each alternative impose?

  • With respect to the spread of CWD, in addition to the requirements established by the APHIS CWD rule, what additional safeguards do states need to mitigate or reduce the risk of disease transmission and why are they needed?

  • What practical or operational problems may be expected from the final rule and from the alternatives suggested by the petitions? How could they be alleviated; and

  • Are there any alternatives that could address the petitioners’ concerns, other than allowing the movement requirements of individual states to take precedence over the federal standard?

The notice requesting comments on the petition is scheduled for publication in the Nov. 3 Federal Register.

Consideration will be give to comments received on or before Dec. 4. Send an original and three copies of postal or commercial delivery comments to Docket APHIS-2006-0118, Regulatory Analysis and Development, PPD, APHIS, Station 3A-03.8, 4700 River Road, Unit 118, Riverdale, Md. 20737-1238. If you wish to submit a comment using the Internet go to the

Federal eRulemaking portal, select “Animal and Plant Health Inspection Service” from the agency drop-down menu; then click on “Submit.” In the Docket ID column, select APHIS-2006-0118 to submit or view public comments and to view supporting and related materials available electronically.

Comments are posted on the Regulations.gov Web site and may also be reviewed at USDA, Room 1141, South Building, 14th St. and Independence Ave., S.W., Washington, D.C., between 8 a.m. and 4:30 p.m., Monday through Friday, excluding holidays. To facilitate entry into the comment reading room, please call (202) 690-2817.

Public Comments Solicited on CWD Rule

The final rule on Chronic Wasting Disease (CWD) Herd Certification Program and Interstate Movement of Farmed or Captive Deer, Elk, and Moose was published in the Federal Register by the U.S. Department of Agriculture, Animal Plant and Health Inspection Service (APHIS) on July 21, 2006 (SCWDS BRIEFS, Vol. 22, No. 2).

The rule was the result of efforts that go back at least to 1998 when a model program for CWD surveillance, control, and eradication was presented to the United States Animal Health Association (USAHA). The final rule was to be implemented on October 19, 2006; however, on September 8, 2006, APHIS announced a delay in the effective date of the rule (Federal Register Vol. 71, No. 174, p. 52983). On November 3, 2006, APHIS announced that it was soliciting public comments on three petitions it had received concerning the final rule and will consider all comments it receives on or before December 4, 2006 (Federal Register, Vol. 71, No. 213, pp. 64650-64651).

The announcement on November 3, and accompanying press release from APHIS stated, “We recently received three petitions requesting a delay in the effective date of the CWD rule and reconsideration of several requirements of the rule. We are currently evaluating the merits of these petitions, and…are making the petitions available for public review and requesting comments on them.” The petitions are from the Association of Fish and Wildlife Agencies, National Assembly of State Animal Health Officials, and USAHA.

APHIS invites the public to comment on any of the issues raised by the petitions, but is particularly interested in receiving comments related to the following areas:

  • Consider the alternatives of implementing a federal interstate movement standard versus allowing individual state standards to apply. What hardships or benefits would each alternative impose?
  • With respect to the spread of CWD, in addition to the requirements established by the APHIS CWD rule, what additional safeguards do states need to mitigate or reduce the risk of disease transmission and why are they needed?
  • What practical or operational problems may be expected from the final rule and from the alternatives suggested by the petitions? How could they be alleviated; and
  • Are there any alternatives that could address the petitioners’ concerns, other than allowing the movement requirements of individual states to take precedence over the federal standard?

The petitions may be viewed and comments may be submitted electronically by going to www.regulations.gov, then select “Animal and Plant Health Inspection Service” from the agency drop-down menu, then click on “Submit.” In the Docket ID column, select APHIS-2006-0118 to submit or view public comments and to see supporting and related materials. Comments also may be reviewed at USDA, Room 1141, South Building, 14th St. and Independence Ave., S.W., Washington, DC, between 8 a.m. and 4:30 p.m., Monday through Friday, excluding holidays. To facilitate entry into the comment reading room, please call (202) 690-2817. Following evaluation of the comments, APHIS will announce the future direction of the federal CWD program. (Prepared by John Fischer)